@conference {40543,
	title = {Brief Communication - Time and incidence of foetal mortality in alpacas},
	booktitle = {Proceedings of the New Zealand Society of Animal Production},
	volume = {53},
	year = {1993},
	month = {Jan},
	pages = {437-438},
	publisher = {New Zealand Society of Animal Production},
	organization = {New Zealand Society of Animal Production},
	abstract = {The deficiency of uridine monophosphate synthase is a disorder of  the de novo pathway for pyrimidine nucleotide biosynthesis. Inherited as an  autosomal recessive, it results in early embryonic mortality for the homozygous  recessive genotype. In the mid-1980{\textquoteright}s, heterozygotes were estimated at 1 - 2\%  among US Holstein cattle. The institution of a testing program for carriers and  refusal by US artificial insemination companies to accept carriers into their  progeny testing programs has eliminated the condition from the top 400 US  Holstein bulls. Bovine citrullinaemia is a defect of the urea cycle. Also  inherited as an autosomal recessive, it results in early postnatal mortality for  the homozygous recessive genotype. In 1990, heterozygotes constituted 0.3\% of  US Holstein cattle. At present, no known carriers are among the top 400 US  Holstein bulls. Carriers for either condition can be detected by convenient  and inexpensive DNA based testing. To guard against these conditions  reappearing, a modest testing program among prominent US bulls should be  continued and importers should insist that imported Holstein germplasm have  no evidence of these conditions.  },
	issn = {1176-5283},
	author = {Ridland M and Knight TW and Wyeth TK and Death AF}
}
