Abstract
The molecular mechanisms involved in involution of the bovine udder, a process which underpins lactation persistency, are poorly understood. During rodent lactation, STAT5 is activated in response to prolactin (PRL) to stimulate milk protein production (Liu et al., 1997). At the onset of involution, STAT5 is rapidly inactivated (Schmitt-Ney et al., 1992), and STAT3 is activated by leukaemia inhibitory factor (LIF) to... Continued
Proceedings of the New Zealand Society of Animal Production, Volume 69, Christchurch, 65-67, 2009
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